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However, the buffering capacity becomes limited as impairment of SAT expansion causes IR [22, 23, 24] as the excess fat are deposited in the visceral adipose tissue (VAT) as well as ectopically in the skeletal muscle, liver, kidney, and heart tissues [25]
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Kreymann B, Ghatei M, Williams G, Bloom S: Glucagon-like peptide-1 736: a physiological incretin in man
In 1987, her paper with Habener and Daniel Drucker showed that GLP-1 stimulates glucose-dependent insulin secretion in the pancreas, suppresses glucagon release, and slows gastric emptying, all of which stabilize blood sugar levels after eating
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